egfr sirna (Santa Cruz Biotechnology)
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Santa Cruz Biotechnology
egfr sirna
Egfr Sirna, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 112 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/egfr+sirna/EGFR+siRNA/pm41326761-342-7-9
Average 93 stars, based on 112 article reviews
Egfr Sirna, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 112 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/egfr+sirna/EGFR+siRNA/pm41326761-342-7-9
Average 93 stars, based on 112 article reviews
egfr sirna - by Bioz Stars,
2026-09
93/100 stars
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Transfection:Article Title: Molecular mechanism of the uptake and toxicity of EGF-LipoAgNPs in EGFR-overexpressing cancer cells. Article Snippet: The High-Capacity cDNA Reverse Transcription Kit, TaqMan probes, and primers corresponding to the genes encoding ACTB (Hs01060665_g1), EGFR (Hs06634166_s1), SHH (Hs01037584_m1), ATM (Hs01037584_m1), CAV1 (Hs01037584_m1), CLTC (Hs01037584_m1), SH3GLB1 (Hs01037584_m1), AP2M1 (Hs01037584_m1), and NFE2L2 (Hs00765730_m1) were purchased from ThermoFisher (Grand Island, USA). .. Article Title: Polymorphic Membrane Protein 17G of Chlamydia psittaci Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host. Article Snippet: .. C. psittaci Infection To identify the effect ofEGFRonC.psittaci6BCinfection,HeLa229 cells were pretreated with different concentrations of cetuximab (Sigma-Aldrich, Shanghai, China), an anti-EGFR antibody, at 37°C for 2 h. Then, the unbound antibodies were removed from the reaction followed by washing 5 times with PBS, and finally, cell cultures were infected with C. psittaci 6BC at an MOI of 5 and incubated at 37°C for 36-48 h. In another experiment, EGFR in HeLa 229 cells was knocked down via transfection with Article Title: N-phenylquinazolin-4-amine-based EGFR TKIs suppress pulmonary fibrosis by modulating the EGFR/ERBB3 axis in epithelial–macrophage interaction Article Snippet: .. Lipofectamine 2000 (Invitrogen; #11668019) was used as a transfection reagent, and Article Title: Collectin-11 promotes fibroblast proliferation and modulates their activation status and extracellular matrix synthesis Article Snippet: Biotinylated BSA (Bio-BSA) was from Beijing Psaitong Biotechnology. .. BSA and L-fucose were sourced from Sigma-Aldrich, Article Title: N-phenylquinazolin-4-amine-based EGFR TKIs suppress pulmonary fibrosis by modulating the EGFR/ERBB3 axis in epithelial-macrophage interaction. Article Snippet: .. Communications Biology | (2025) 8:1723 12 and Infection:Article Title: Polymorphic Membrane Protein 17G of Chlamydia psittaci Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host. Article Snippet: .. C. psittaci Infection To identify the effect ofEGFRonC.psittaci6BCinfection,HeLa229 cells were pretreated with different concentrations of cetuximab (Sigma-Aldrich, Shanghai, China), an anti-EGFR antibody, at 37°C for 2 h. Then, the unbound antibodies were removed from the reaction followed by washing 5 times with PBS, and finally, cell cultures were infected with C. psittaci 6BC at an MOI of 5 and incubated at 37°C for 36-48 h. In another experiment, EGFR in HeLa 229 cells was knocked down via transfection with Incubation:Article Title: Polymorphic Membrane Protein 17G of Chlamydia psittaci Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host. Article Snippet: .. C. psittaci Infection To identify the effect ofEGFRonC.psittaci6BCinfection,HeLa229 cells were pretreated with different concentrations of cetuximab (Sigma-Aldrich, Shanghai, China), an anti-EGFR antibody, at 37°C for 2 h. Then, the unbound antibodies were removed from the reaction followed by washing 5 times with PBS, and finally, cell cultures were infected with C. psittaci 6BC at an MOI of 5 and incubated at 37°C for 36-48 h. In another experiment, EGFR in HeLa 229 cells was knocked down via transfection with Knockdown:Article Title: Polymorphic Membrane Protein 17G of Chlamydia psittaci Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host. Article Snippet: .. C. psittaci Infection To identify the effect ofEGFRonC.psittaci6BCinfection,HeLa229 cells were pretreated with different concentrations of cetuximab (Sigma-Aldrich, Shanghai, China), an anti-EGFR antibody, at 37°C for 2 h. Then, the unbound antibodies were removed from the reaction followed by washing 5 times with PBS, and finally, cell cultures were infected with C. psittaci 6BC at an MOI of 5 and incubated at 37°C for 36-48 h. In another experiment, EGFR in HeLa 229 cells was knocked down via transfection with Western Blot:Article Title: Polymorphic Membrane Protein 17G of Chlamydia psittaci Mediated the Binding and Invasion of Bacteria to Host Cells by Interacting and Activating EGFR of the Host. Article Snippet: .. C. psittaci Infection To identify the effect ofEGFRonC.psittaci6BCinfection,HeLa229 cells were pretreated with different concentrations of cetuximab (Sigma-Aldrich, Shanghai, China), an anti-EGFR antibody, at 37°C for 2 h. Then, the unbound antibodies were removed from the reaction followed by washing 5 times with PBS, and finally, cell cultures were infected with C. psittaci 6BC at an MOI of 5 and incubated at 37°C for 36-48 h. In another experiment, EGFR in HeLa 229 cells was knocked down via transfection with Expressing:Article Title: Tyrosine sulphation of CXCR4 induces the migration of fibroblast in OSF. Article Snippet: Oral submucous fibrosis (OSF) is a potentially cancerous oral mucosal disease, which is generally considered to be caused by areca nut chewing and characterised by progressive accumulation of collagen fibres in the lamina propria and the underlying submucosal layer (Shruti et al., 2009).. Fibrotic remodelling is a common pathological abnormality in OSF development, while fibroblasts (FBs) are the key cell type for the pathological process (Isaac et al., 2008).. In fibrotic progression, FBs can be induced to migrate and recruit into the provisional matrix, followed by deposition and production of excessive extracellular matrix under inflammatory stimulation. Plasmid Preparation:Article Title: Tyrosine sulphation of CXCR4 induces the migration of fibroblast in OSF. Article Snippet: Oral submucous fibrosis (OSF) is a potentially cancerous oral mucosal disease, which is generally considered to be caused by areca nut chewing and characterised by progressive accumulation of collagen fibres in the lamina propria and the underlying submucosal layer (Shruti et al., 2009).. Fibrotic remodelling is a common pathological abnormality in OSF development, while fibroblasts (FBs) are the key cell type for the pathological process (Isaac et al., 2008).. In fibrotic progression, FBs can be induced to migrate and recruit into the provisional matrix, followed by deposition and production of excessive extracellular matrix under inflammatory stimulation. Construct:Article Title: Tyrosine sulphation of CXCR4 induces the migration of fibroblast in OSF. Article Snippet: Oral submucous fibrosis (OSF) is a potentially cancerous oral mucosal disease, which is generally considered to be caused by areca nut chewing and characterised by progressive accumulation of collagen fibres in the lamina propria and the underlying submucosal layer (Shruti et al., 2009).. Fibrotic remodelling is a common pathological abnormality in OSF development, while fibroblasts (FBs) are the key cell type for the pathological process (Isaac et al., 2008).. In fibrotic progression, FBs can be induced to migrate and recruit into the provisional matrix, followed by deposition and production of excessive extracellular matrix under inflammatory stimulation. Cloning:Article Title: Tyrosine sulphation of CXCR4 induces the migration of fibroblast in OSF. Article Snippet: Oral submucous fibrosis (OSF) is a potentially cancerous oral mucosal disease, which is generally considered to be caused by areca nut chewing and characterised by progressive accumulation of collagen fibres in the lamina propria and the underlying submucosal layer (Shruti et al., 2009).. Fibrotic remodelling is a common pathological abnormality in OSF development, while fibroblasts (FBs) are the key cell type for the pathological process (Isaac et al., 2008).. In fibrotic progression, FBs can be induced to migrate and recruit into the provisional matrix, followed by deposition and production of excessive extracellular matrix under inflammatory stimulation. Control:Article Title: Glycopolymer-Cell-Penetrating Peptide (CPP) Conjugates for Efficient Epidermal Growth Factor Receptor (EGFR) Silencing. Article Snippet: .. Control, fluorescein isothiocyanate FITC-conjugated control siRNA, Article Title: N-phenylquinazolin-4-amine-based EGFR TKIs suppress pulmonary fibrosis by modulating the EGFR/ERBB3 axis in epithelial–macrophage interaction Article Snippet: .. Lipofectamine 2000 (Invitrogen; #11668019) was used as a transfection reagent, and Article Title: Collectin-11 promotes fibroblast proliferation and modulates their activation status and extracellular matrix synthesis Article Snippet: Biotinylated BSA (Bio-BSA) was from Beijing Psaitong Biotechnology. .. BSA and L-fucose were sourced from Sigma-Aldrich, Article Title: N-phenylquinazolin-4-amine-based EGFR TKIs suppress pulmonary fibrosis by modulating the EGFR/ERBB3 axis in epithelial-macrophage interaction. Article Snippet: .. Communications Biology | (2025) 8:1723 12 and |
![Knockdown of epidermal growth factor receptor blocked the positive effects of epiregulin on migration, chemotaxis, and osteogenic potential of mouse bone marrow stem cells under inflammatory conditions. A: Quantitative analysis of quantitative real-time polymerase chain reaction showed that the transcription of epidermal growth factor receptor ( <t>Egfr</t> ) mRNA was decreased in the EGFR single hairpin <t>RNA</t> <t>(shRNA)</t> group ( n = 3, Student’s t -test). Glyceraldehyde 3-phosphate dehydrogenase ( Gapdh ) was used as an internal control; B: Expression of EGFR was revealed in mouse bone marrow stem cells by western blotting. GAPDH was used as the internal control; C and D: Scratch-simulated wound migration assay results ( n = 3, one-way analysis of variance [ANOVA]). Scale bar: 500 μm; E and F: Transwell chemotaxis assay results ( n = 3, one-way ANOVA). Scale bar: 50 μm; G: Alizarin Red staining; H: Expression of osterix (OSX) and osteocalcin (OCN) was revealed in mouse bone marrow stem cells that were cultured in the inflammatory condition by western blotting. Error bars represent standard deviation. a P ≤ 0.05; b P ≤ 0.01. EREG: Epiregulin.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_8805/pmc12968805/pmc12968805__wjsc-18-2-114032-g002.jpg)
![Epiregulin activated the epidermal growth factor receptor-extracellular signal-regulated kinase 1/2 signaling pathway to promote migration, chemotaxis, and osteogenic differentiation of mouse bone marrow stem cells under inflammatory conditions. A: Expression of mitogen-activated protein kinase signaling pathway were revealed in mouse bone marrow stem cells (mBMSCs) that were cultured in the inflammatory condition by western blotting. β-actin was used as the internal control; B: Expression of phosphorylated extracellular signal-regulated kinase (p-ERK) and ERK were detected in mBMSCs that were cultured in the inflammation condition by western blotting; C and D: Scratch-simulated wound migration assay results ( n = 3, one-way analysis of variance [ANOVA]). Scale bar: 500 μm; E and F: Transwell chemotaxis assay results ( n = 3, one-way ANOVA). Scale bar: 100 μm; G: Alizarin red staining; H: Expression of osterix (OSX) and osteocalcin (OCN) were revealed in mBMSCs that were cultured in the inflammatory condition by western blotting. Error bars represent standard deviation ( n = 3). a P ≤ 0.05; b P ≤ 0.01. EGFR: Epidermal growth factor receptor; EREG: Epiregulin; JNK: C-Jun N-terminal kinase; MAPK: Mitogen-activated protein kinase.](https://pub-med-central-images-cdn.bioz.com/pub_med_central_ids_ending_with_8805/pmc12968805/pmc12968805__wjsc-18-2-114032-g003.jpg)
